When traveling or socializing we have less control of our food quality and preparation. What Carnivore options do you choose when your out?

    • jet@hackertalks.comOPMEnglish
      1·
      2 days ago

      well, yes, I’m trying to avoid elevated blood glucose, and industrial oils. That is the point of the community.

      Carnivore makes it very simple for me to maintain my health plan.

        • jet@hackertalks.comOPMEnglish
          1·
          2 days ago

          If your asking the more general question about the difference between seed oils and animal fats -

          Animal fats are saturated, and have a high omega 3 content. Animal fats do not interfere with the bodies cholesterol and are not associated with insulin resistance

          • ProfessorOwl_PhD [any]@hexbear.netEnglish
            3·
            1 day ago

            Ok, I think you’ve gotten a bit confused by someone peddling a scam diet.

            • Basically all fat sources are a mix of saturated and unsaturated fats
            • Coconut and palm oil are much better sources of saturated fats than animal fat, but you don’t really want to be consuming those either, because
            • Saturated fats have definitely been associated with insulin resistance - monounsaturated fats are the one that reduce it.

            Canola and Olive oils are the ones you want to be using to help with cholesterol and insulin resistance.

            • jet@hackertalks.comOPMEnglish
              11·
              1 day ago

              Can you please cite direct sources? All the papers I’ve read indicate the opposite

              Saturated fats have definitely been associated with insulin resistance

              What is the mechanism is this? How does fat increase insulin resistance? Fat does not trigger a insulin response.

              • ProfessorOwl_PhD [any]@hexbear.netEnglish
                2·
                1 day ago

                Well the most famous I know of is the Kanwu Study

                How does fat increase insulin resistance? Fat does not trigger a insulin response.

                Insulin regulates your metabolism and the conversion between sugars and fats within your body, affecting everything from sugar production to protein synthesis. Your body isn’t a simple mechanism where everything has one job and is unaffected by the rest of the chemical soup. Fats don’t trigger a release of insulin, that doesn’t mean they have no effect on it.

                • jet@hackertalks.comOPMEnglish
                  1·
                  1 day ago

                  Substituting dietary saturated for monounsaturated fat impairs insulin sensitivity

                  Thank you for the reference, it was a interesting read. This study has the Saturated fat group at 50% saturated fat, and 50% unsaturated fats, its not a very clean signal. I’m not sure why the SAFA group had a reduced response to a GTT, it warrants further study.

                  Since the study you reference is from 2001, there are more recent studies that reference it.

                  https://www.sciencedirect.com/science/article/pii/S2161831323000674

                  Saturated Fatty Acid Intake and Risk of Type 2 Diabetes: An Updated Systematic Review and Dose–Response Meta-Analysis of Cohort Studies

                  There was evidence of publication bias among studies on dietary total SFAs and T2DM. Our results indicated no significant association between dietary total SFA and risk of T2DM.

                  Some of the experimental studies support the notion that dietary fats, and SFAs in particular, are associated with the development of insulin resistance and type 2 diabetes mellitus (T2DM) (2–5). On the other hand, the majority of more recent cohort studies have indicated no association between dietary SFAs and the incidence of T2DM

                  The linear dose–response meta-analysis of the main 13 studies showed no linear association between increasing intake of SFAs and T2DM risk (HR: 0.93; 95% CI: 0.84, 1.03). From 13 cohort studies regarding the association between total SFAs and T2DM risk, 7 studies (5–7, 9, 31, 33, 34) reported sufficient data for the nonlinear doseresponse analyses. There was no evidence of a U- or J-shaped association between total SFA intake and risk of T2DM (Pnonlinearity = 0.153; n = 7; Figure 3). Supplemental Figure 5 presents the results for publication bias. Overall, there was evidence of publication bias with the Egger test (P = 0.032).