That's just *56. Amyloid has been known for decades to play some role, even though *56's data was fraudulent (for a lay-friendly discussion, see https://www.alzheimers.org.uk/what-we-do/researchers/news/explaining-amyloid-research-study-controversy). Amyloid is certainly not the only thing at play, but it does play some role.
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Yes, it plays a role. What exactly it's doing is unclear, and it's probably more that it's setting up tau to do the real nasty stuff, but it contributes. We know that from experimental work in nonhuman animal models and converging longitudinal work in humans. See, for example: https://www.cell.com/neuron/pdfExtended/S0896-6273(22)00305-1